“These results suggest that C. militaris inhibited ovarian cancer cell proliferation, survival, and migration, possibly through the coordination between TNF-α/TNFR1 signaling and NF-κB activation. Taken together, our findings provide a new insight into a novel treatment strategy for ovarian cancer using C. militaris.”
https://bmccomplementmedtherapies.biomedcentral.com/articles/10.1186/s12906-019-2780-5
