Honey has almost equal or slightly superior effects when compared with conventional treatments for acute wounds and superficial partial thickness burns.
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Venom from honeybees has been found to rapidly kill aggressive and hard-to-treat breast cancer cells, according to potentially groundbreaking new Australian research.
“In recent years, natural products, which originate from plants, animals, and fungi, together with their bioactive compounds have been intensively explored and studied for their therapeutic potentials for various diseases such as cardiovascular, diabetes, hypertension, reproductive, cancer, and neurodegenerative diseases. Neurodegenerative diseases, including Alzheimer’s disease, Huntington’s disease, Parkinson’s disease, and amyotrophic lateral sclerosis are characterized by the progressive dysfunction and loss of neuronal structure and function that resulted in the neuronal cell death. Since the multifactorial pathological mechanisms are associated with neurodegeneration, targeting multiple mechanisms of actions and neuroprotection approach, which involves preventing cell death and restoring the function to damaged neurons, could be promising strategies for the prevention and therapeutic of neurodegenerative diseases. Natural products have emerged as potential neuroprotective agents for the treatment of neurodegenerative diseases. This review focused on the therapeutic potential of natural products and their bioactive compounds to exert a neuroprotective effect on the pathologies of neurodegenerative diseases.”
Honey acts as an antibacterial agent against many bacteria (1). There are two sorts of antibacterial agents or so called “inhibines.†One of them is heat- and light-sensitive and has its origin in the H2O2, produced by honey glucose oxidase (2,3,4). Some workers believe that hydrogen peroxide is the main antibacterial agent (2,5,6). Other authors find that the non-peroxide activity is the more important one (7,8,9). The H2O2 amount in honey is very small and it can be produced only after aerobic incubation of diluted honey solutions, which might mean that it is not very important for the antibacterial action of honey (10). The argumentations of the pro and contra peroxide side are based on the results with the specific antibacterial test used. However, a certain antibacterial test might be sensitive only to certain types of antibacterian substances. In a previous study from our laboratory it was found that while in an agar disc diffusion test only the peroxide activity was measured, in a liquid medium test only the non-peroxide substances were active(10).
https://link.springer.com/chapter/10.1007%2F978-1-4757-9371-0_4
https://thisis42.podlink.to/honey
Skin and chronic wound infections caused by highly antibiotic resistant bacteria such as methicillin-resistant Staphylococcus aureus (MRSA) are an increasing and urgent health problem worldwide, particularly with sharp increases in obesity and diabetes. New Zealand manuka honey has potent broad-spectrum antimicrobial activity, has been shown to inhibit the growth of MRSA strains, and bacteria resistant to this honey have not been obtainable in the laboratory. Combinational treatment of chronic wounds with manuka honey and common antibiotics may offer a wide range of advantages including synergistic enhancement of the antibacterial activity, reduction of the effective dose of the antibiotic, and reduction of the risk of antibiotic resistance. The aim of this study was to investigate the effect of Medihoney in combination with the widely used antibiotic rifampicin on S. aureus. Using checkerboard microdilution assays, time-kill curve experiments and agar diffusion assays, we show a synergism between Medihoney and rifampicin against MRSA and clinical isolates of S. aureus. Furthermore, the Medihoney/rifampicin combination stopped the appearance of rifampicin-resistant S. aureus in vitro. Methylglyoxal (MGO), believed to be the major antibacterial compound in manuka honey, did not act synergistically with rifampicin and is therefore not the sole factor responsible for the synergistic effect of manuka honey with rifampicin. Our findings support the idea that a combination of honey and antibiotics may be an effective new antimicrobial therapy for chronic wound infections.
https://www.ncbi.nlm.nih.gov/m/pubmed/23469049/?fbclid=IwAR1w8X7OYE9RRw27zOFFO5zmevpNahNN2pZYbZIz0CoGiysH0bs-0dtZT_4
https://ozhoneyproject.wordpress.com/?fbclid=IwAR0CxSnXUwraq14GBTTMcnkRg2Mzq6vheRQjKwVGjDO0XWf4XU8bGhNgt2w
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4075678/?fbclid=IwAR1jgUas1vYC_cvfXWiBxvw0BhWlOeEGsZMX_msQUoxFd1jt1pvS417BAaU
Deli Bal has a long history considering its human consumption (Mad Honey, Miel Fou, Crazy Honey, Toxic Honey) as aphrodisiac but also a war weapon: Pliny and Strabo confirmed that locals used Deli Bal against the armies of Xenophon in 401 BCE and against Pompey in 69 BCE. When Pompey the Great attacked troops in Turkey they were poisoned because they did not know the effects of Deli Bal. Roman Soldiers ate too much of it and became disoriented leading their army to a defeat. Historically the diterpene grayanotoxin in the honey is produced when bees pollinate flowers of rhododendron ponticum and rhododendron luteum. In the 18th century Deli Bal was added to alcoholic drinks in Europe to provide a quick “wake me up energizer†with stimulants effects. Deli Bal is still deliberately produced in Turkey and Nepal for its medical properties.
http://magic-bees.com/shop/content/6-deli-bal-buy-mad-honey